Study of Genetic Anomalies of Complement Related Proteins in Patients With IgA Glomerulonephritis

Completed

Conditions studied: IGA Glomerulonephritis

In brief

OBJECTIVES: I. Determine whether allelic differences associated with the fourth component of complement, type-1 complement receptor expressed on erythrocytes, and Fc receptor FcgRIII contribute to the pathogenesis of IgA glomerulonephritis (IgA-N). II. Compare genetic anomalies of these key components in immune complex processing and clearance between juvenile vs adult onset IgA-N vs normal controls.

Key facts

Study ID

NCT00004305

Run by

National Center for Research Resources (NCRR)

People needed

105

Starts

1998-01-01

Last updated by the study team

2005-06-24

Who can join

Age: any. Sex: any. Healthy volunteers: not accepted.

Where it is running

• Ohio State University — Columbus, Ohio, United States

Full record on ClinicalTrials.gov

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